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《Nature Genetics》杂志2017年发表文章

  • 创建者:Science
  • 创建时间:2017-01-03 15:03
  • 修改时间:2018-02-26 21:16
  • 介绍:Journal:Nature Genetics<br/>Year:2017
  • 关键词:
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ng.3771-A somatic-mutational process recurrently duplicates germline susceptibility loci and tissue-specific super-enhancers in breast cancers11 p
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  • 2017-01-24 08:49
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ng.3772-Pediatric non–Down syndrome acute megakaryoblastic leukemia is characterized by distinct genomic subsets with varying outcomes8 p
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ng.3773-Acquired CYP19A1 amplification is an early specific mechanism of aromatase inhibitor resistance in ERα metastatic breast cancer9 p
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ng.3753-Epigenomic reprogramming during pancreatic cancer progression links anabolic glucose metabolism to distant metastasis12 p
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  • 2017-01-17 08:40
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ng.3756-Precision oncology for acute myeloid leukemia using a knowledge bank approach10 p
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  • 2017-01-17 08:40
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ng.3762-Biallelic mutations in the 3′ exonuclease TOE1 cause pontocerebellar hypoplasia and uncover a role in snRNA processing10 p
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ng.3764-Limited heterogeneity of known driver gene mutations among the metastases of individual patients with pancreatic cancer11 p
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  • 2017-01-17 08:39
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ng.3766-Case–control association mapping by proxy using family history of disease9 p
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ng.3761-Exploring the genetic architecture of inflammatory bowel disease by whole-genome sequencing identifies association at ADCY79 p
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  • 2017-01-10 09:11
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ng.3765-De novo mutations in SMCHD1 cause Bosma arhinia microphthalmia syndrome and abrogate nasal development9 p
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ng.3760-Genome-wide association study implicates immune activation of multiple integrin genes in inflammatory bowel disease8 p
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ng.3755-Genome-wide association study identifies distinct genetic contributions to prognosis and susceptibility in Crohn´s disease9 p
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ng.3743-SMCHD1 mutations associated with a rare muscular dystrophy can also cause isolated arhinia and Bosma arhinia microphthalmia syndrome14 p
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ng.3757-Impaired H3K36 methylation defines a subset of head and neck squamous cell carcinomas8 p
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ng.3711-Qki deficiency maintains stemness of glioma stem cells in suboptimal environment by downregulating endolysosomal degradation15 p
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